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Jonathan R Silva

Targeting the IKs Channel PKA Phosphorylation Axis to Restore Its Function in High-Risk LQT1 Variants
Conformations of voltage-sensing domain III differentially define NaV channel closed- and open-state inactivation
Modulation of the effects of class Ib antiarrhythmics on cardiac NaV1.5-encoded channels by accessory NaVβ subunits
Molecular pathology of sodium channel beta-subunit variants
Gating control of the cardiac sodium channel Nav1.5 by its β3-subunit involves distinct roles for a transmembrane glutamic acid and the extracellular domain
Mechanisms and models of cardiac sodium channel inactivation
Mechanisms of noncovalent β subunit regulation of NaV channel gating
Regulation of Na+ channel inactivation by the DIII and DIV voltage-sensing domains
A novel NaV1.5 voltage sensor mutation associated with severe atrial and ventricular arrhythmias
Molecular motions that shape the cardiac action potential : Insights from voltage clamp fl uorometry
Direct measurement of cardiac Na+ channel conformations reveals molecular pathologies of inherited mutations

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